THE PLATFORM

IDENTIFY AND TREAT YOUR ALZHEIMER'S SYMPTOMS

What is

Alzheimer’s disease is one of the most significant health care problems nationally and globally. Recently, the first description of the reversal of cognitive decline in patients with early Alzheimer’s disease or its precursors, MCI (mild cognitive impairment) and SCI (subjective cognitive impairment), was published. The therapeutic approach used was programmatic and personalized rather than monotherapeutic and invariant, and was dubbed metabolic enhancement for neuro degeneration (MEND). Patients who had to discontinue work were able to return to work, and those struggling at work were able to improve their performance.

ReCODE is currently available in the US for $1,399. The cost includes all necessary assessments, blood labs, genome tests, and access to a Trained Practitioner Locator Tool. For further questions, contact us at support@ahnphealth.com. If you are located outside of the US, sign up to be notified when ReCODE is available in your area.

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THE REPORT

The ReCODE engine generates a report for you and your physician. We have created a new category of medical information and analysis where we take highly complex sets of biometric and genetic data sets to generate medical information and a simple image of YOU for you and your physician.

Partnering with Dr. Dale Bredesen, Alzheimer’s research pioneer and inventor of the MEND protocol (now superseded by the more sophisticated ReCODE protocol), AHNP Precision Health has created the first software product that identifies subtypes of cognitive decline (types 1-3 Alzheimer’s disease, published by Dr. Bredesen). The report delivers optimal personalized medical information that allows users and practitioners to achieve unprecedented success in the prevention and reversal of cognitive decline. The report is generated from a very specific set of lab and genome tests curated by Dr. Bredesen.

The ReCODE Report costs $1,399, which includes all necessary assessments, blood labs, genome tests, and access to a Trained Practitioner Locator Tool. For further questions, contact us at support@ahnphealth.com.

WHY

  • Identify the subtypes and source of your cognitive decline.
  • ReCODE is the first to report the reversal of cognitive decline in patients with early Alzheimer's disease.
  • An affordable solution to reverse the symptoms of Alzheimer' disease and identify preventative solutions.
  • Includes all metabolic and genetic laboratory assessment to generate a personalized ReCODE report.
  • Access to a directory of ReCODE trained practitioners, coaches, and nutritionists.
  • ReCODE is currently available in the US. If you are located outside of the US, sign up to be notified when ReCODE is available in your area.
FAQ

How do I get a Report

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THE SCIENCE OF

Keep up to date with Dr. Bredesen's latest research

Reversal of Cognitive Decline in Alzheimer’s Disease
Alzheimer’s disease is one of the most significant healthcare problems nationally and globally. Recently, the first description of the reversal of cognitive decline in patients with early Alzheimer’s disease or its precursors, MCI (mild cognitive impairment) and SCI (subjective cognitive impairment), was published.  The therapeutic approach used was programmatic and personalized rather than monotherapeutic and invariant, and was dubbed metabolic enhancement for neurodegeneration (MEND). Patients who had to discontinue work were able to return to work, and those struggling at work were able to improve their performance. The patients, their spouses, and their co‐workers all reported clear improvements. READ MORE


Transcriptional Effects of ApoE4: Relevance to Alzheimer's Disease
The major genetic risk factor for sporadic Alzheimer's disease (AD) is the lipid binding and transporting carrier protein apolipoprotein E, epsilon 4 allele (ApoE4). One of the unsolved mysteries of AD is how the presence of ApoE4 elicits this age-associated, currently incurable neurodegenerative disease. Recently, we showed that ApoE4 acts as a transcription factor and binds to the promoters of genes involved in a range of processes linked to aging and AD disease pathogenesis. READ MORE

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